Insulin resistance and diabetes mellitus type 2 develop when there Blood Sugar Formula Review  is an imbalance between conditions that promote excessive fatty acid synthesis combined with deficient fatty acid oxidation. The conditions mentioned are such things as dietary, genetic, hormonal, and exercise related areas. Of most concern in this paper, is the influence that genetic modifications may have on fatty acid metabolism. Much research has been done in this area, especially on the mouse, whose genome is considerably similar to the human genome. Through the study of the mouse genome, advances in human health, in the way of better insulin sensitivity, can be realized.

There are two main mouse models that illustrate which genes are involved most in fatty acid metabolism. The first model deals with mouse mutants used to study excessive and deficient fatty acid synthesis and oxidation. In regards to fatty acid/triacylgylcerol synthesis, sterol-response element binding protein over-expression can cause trigger an excess in these compounds. If one carries the diacylglycerol transferase -/- gene or the short-chain acyl coenzyme A dehydrogenase -/- gene, deficient level of fatty acid synthesis will occur. In regards to fatty acid oxidation, acetyl coenzyme A carboxylase 2 -/- gene or peroxisomal proliferator-activated receptor gene over-expression can result in a very high oxidation rate of fatty acids from the blood stream. If very longchain coenzyme A dehydrogenase -/- gene or long-chain acyl coenzyme A dehydrogenase -/- gene are part of one's genome, fatty acid oxidation in the body will be deficient.
 

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